Conversely, other studies report a correlation between high HCC-FABP4 expression, recurrence-free survival in HCC patients with underlying HBV, and FABP4-dependent inhibition of cell proliferation in HBV+ cells in vitro [195], indicating that the underlying pathology impacts the role of FABP4 in hepatic tumor progression. The gene discussed is FABP4; the disease is hepatocellular carcinoma.