In response to viral or fungal stimuli, airway epithelial cells from individuals with asthma release even greater amounts of TSLP, [9,10,11,12] thereby polarizing T helper 2 (Th2) cells and expanding innate lymphoid (ILC2) cells to enhance the production of T2 cytokines, including IL-4, IL-13, and IL-5 [13,14,15]. This evidence concerns the gene TSLP and asthma.