Analysis of blood samples collected during reperfusion (TR) showed an abundant synthesis and release of MMP-2 (2048.0 ± 419.0 ng/mL) and MMP-9 (165.1 ± 19.07 mg/mL) in the MI group, followed by increased degradation and extracellular release of cardiac proteins such as MLC1 (340.7 ± 155.7) and BNP-26 (0.16 ± 0.064 mg/mL) (Figure 8). The gene discussed is MMP9; the disease is myocardial infarction.