Overall, this suggests that at a transcriptional level, the upregulation of cd36 together with the downregulation of lpl, fatp1 and fabp1, as observed in the HF+HT_ST group, agrees with a protective role of HT in the liver only under a lipid overload context, and for the same reason, this effect was not observed when the additive was given to the restricted group. This evidence concerns the gene CD36 and hydrops fetalis.