Overall, this suggests that at a transcriptional level, the upregulation of cd36 together with the downregulation of lpl, fatp1 and fabp1, as observed in the HF+HT_ST group, agrees with a protective role of HT in the liver only under a lipid overload context, and for the same reason, this effect was not observed when the additive was given to the restricted group. Here, LPL is linked to hydrops fetalis.