EGR1 played an important role in myocardial ischemia/reperfusion injury.[23] EGR1 participated in the regulation of inflammation and fibrosis in postinfarcted hearts.[24] Oxidative stress-induced EGR1 was involved in cardiac infarction, and it was able to effectively suppress ventricular arrhythmias in postinfarcted hearts by regulating EGR1 expression, which provided a new therapeutic strategy for ischemic arrhythmias in the clinic.[25]. The gene discussed is EGR1; the disease is Ventricular arrhythmia.