Therefore, in the present study, through the exploration of the machinery related to the maintenance of mitochondrial homeostasis (including mitochondrial fission, fusion, transport, and biogenesis) in a cellular model of UQCRC1 parkinsonism, the therapeutic potential of the targeting of mitochondrial dynamics by formoterol for the future therapy of PD was examined. The gene discussed is UQCRC1; the disease is Parkinson disease.