F11R and juvenile Huntington disease: GO results performed with Genecodis showed that miR156e putative targets are enriched in biological processes that include stress-activated protein kinase signalling cascade (CCDC88C), memory T cell extravasation, and regulation of membrane permeability and bicellular tight junction assembly (F11R), (Table 2), and the Huntington disease pathway (ARL4C and ARF3) (Table 5).