Thus,CPs protect cells from reactive nitrogen and oxygen species.22,24 However, the role of CPs in the aggregation of amyloidogenic proteinsis unclear.19−21 Recent studies from our group showed that CPs alteredthe aggregation rate of α-synuclein, a small membrane proteinthat is linked to Parkinson’s disease.25 However, no changes in the toxicity of fibrils formed in the presenceand absence of CPs by α-synuclein were observed. The gene discussed is CAD; the disease is Parkinson disease.