After intervention with FA, a series of clinical indices of DKD mice showed improvement, wherein the expression of LC3 was upregulated and the expression of p62 and interleukin-1β (IL-1β) were inhibited, confirming that FA plays a therapeutic role in DKD by increasing autophagy and inhibiting inflammation (Ma et al., 2022). Here, MAP1LC3A is linked to diabetic kidney disease.