Clonogenic assays also unveiled that the knockdown of RAD51 partially reversed the increased clonogenic capacity of cells observed after circCDYL2 overexpression, whereas the expression of RAD51 enhanced the survival of nasopharyngeal carcinoma cells following circCDYL2 knockdown (Fig. 4C, Supplemental Fig. 6A). The gene discussed is RAD51; the disease is nasopharyngeal carcinoma.