Based on the findings of KEGG enrichment analysis, it was observed that the high-risk group exhibited significant enrichment of differential genes primarily associated with pathways related to cell proliferation (cell cycle), pro-inflammatory processes (cytokine-cytokine receptor interaction, TNF signaling pathway), and pro-tumor mechanisms (Hippo signal pathway, IL-17 signaling pathway) [50–52]. The gene discussed is TNF; the disease is neoplasm.