To assess the inflammatory milieu within the ARDS model, total cell counts in BALF were performed, showing a substantial increase in cellularity in ARDS rats, which was significantly reduced following AAV-Fcgr2b intervention (Fig. 3E).Subsequently, we utilized immunofluorescence to detect the fluorescence intensity of the apoptosis marker Cleaved-Cas-3 in lung tissue. This evidence concerns the gene FCGR2B and acute respiratory distress syndrome.