Consistent with the results in PBC patients and HBV-infected patients, there were positively correlations between both collagen+ and KRT19+ areas (r = 0.724, p = 0.001), as well as α-SMA+ and KRT19+ areas in NAFLD patients with fibrotic progression (r = 0.619, p = 0.006) (Figure 1B-D, Figure S1B). This evidence concerns the gene KRT19 and metabolic dysfunction-associated steatotic liver disease.