CEBPB and Alzheimer disease: Furthermore, immunohistochemical studies have validated robust colocalization between C/EBPβ‐immunoreactive microglia and Aβ deposits and Aβ immunoreactive microvessels, along with pronounced immunoreactivity in pathologically vulnerable regions of the AD brain, contrasting with comparatively faint staining in corresponding areas of the non‐demented (ND) brain or pathologically spared regions of the AD brain (e.g., cerebellum).31