Likewise, while miR-650 is thought to promote NLRP6 inflammasome-related apoptosis, NLRP6 itself acts as an auto-inhibitor by decreasing the apoptosis increased by the effect of miR-650 through a direct binding of the Nlrp6 3’UTR and miR-650, to restore cell homeostasis after infection clearance [261]. This evidence concerns the gene NLRP6 and infection.