In prior studies, MUC13 overexpression was documented in GC [119], and this dysregulated MUC13 signaling was shown to confer protection against apoptosis in colorectal cancer (CRC) cells through the activation of the NF-κB pathway [120], as well as to facilitate the progression of intrahepatic cholangiocarcinoma by activating the EGFR/PI3K/AKT pathways [121]. The gene discussed is AKT1; the disease is gastric cancer.