We confirmed the interaction between δ‐catenin and β‐TRCP in gastric cancer cells by co‐IP assay (Figure 4H) and demonstrated that M2‐EX treatment attenuated the interaction between δ‐catenin and β‐TRCP while si‐MALAT1 M2‐EX did not have this effect (Figure 4I). The gene discussed is MALAT1; the disease is gastric cancer.