Pushkar et al. showed that MALAT1 enhances the translation of the metabolic transcription factor TCF7L2 by upregulating SRSF1 and activating the mTORC1‐4EBP1 axis, contributing to HCC progression.[38] In multiple myeloma (MM), MALAT1 enhances glycolysis in cancer cells via the regulation of miR‐1271‐5p/SOX13 axis.[39] In this study, we reported that exosomal MALAT1 from M2 TAMs stabilized the δ‐catenin protein in gastric cancer cells by suppressing β‐TRCP‐mediated ubiquitination. This evidence concerns the gene TCF7L2 and hepatocellular carcinoma.