Pushkar et al. showed that MALAT1 enhances the translation of the metabolic transcription factor TCF7L2 by upregulating SRSF1 and activating the mTORC1‐4EBP1 axis, contributing to HCC progression.[38] In multiple myeloma (MM), MALAT1 enhances glycolysis in cancer cells via the regulation of miR‐1271‐5p/SOX13 axis.[39] In this study, we reported that exosomal MALAT1 from M2 TAMs stabilized the δ‐catenin protein in gastric cancer cells by suppressing β‐TRCP‐mediated ubiquitination. Here, TCF7L2 is linked to Miyoshi myopathy.