Exosomes from M2 TAMs deliver miR‐3679‐5p to lung cancer cells, which downregulates the expression of E3 ligase NEDD4L and stabilizes c‐Myc, leading to elevated glycolysis and chemoresistance.[30] Herein, we reported that MALAT1 was enriched in M2 TAM exosomes and transferred from M2 TAMs to gastric cancer cells to induce glycolysis, showing that M2 TAM exosomes may induce cancer glycolysis through distinct cargos. Here, MYC is linked to lung carcinoma.