Genes with cell type-specific expression changes in active EoE that normalized in remission were enriched for immune functions and regulators of immune system processes, exocytosis, and peptidase activity, including multiple genes in fibroblasts, macrophages, and cDC2Bs (Fig. 5b, Supplementary Fig. 6a, Supplementary Data 5, Methods). This evidence concerns the gene LAP3 and eosinophilic esophagitis.