In addition, chemokine (C‐C) motif receptor‐like 2 (Ccrl2), a chemerin receptor with no signaling capabilities, has no effect on lung inflammation induced by exposure to O3 but facilitates inflammatory processes in a mouse model of atopic asthma (Malik et al., 2017; Otero et al., 2010). The gene discussed is CCRL2; the disease is atopic asthma.