JAK/STAT3 signaling has been reported to activate FAO through the transcription of CPT1B in breast cancers.[26] We observed that knockdown of STAT3 in MDA‐MB‐231 cells indeed reduced relative mRNA levels and protein levels of CPT1A and CPT1B (Figure S4C,D, Supporting Information). The gene discussed is CPT1B; the disease is breast carcinoma.