It is also possible that the anti-PYSRA antibody limits Plasmodium invasion of erythrocytes and reduces parasitemia, but the validation of the binding site of SRA protein to the erythrocyte membrane and further animal experiments are needed to verify the possibility of this conjecture, and from the results of the current experiments, it is possible that the anti-PYSRA antibody prevents PYsra from binding to CD68, which reduces the inflammation and alters the outcome of infection in the mice. The gene discussed is CD68; the disease is infection.