Thus, as occurs in human BL tumors, the ΔEBNA2 + Myc lymphomas have a more stringent form of EBV latency (EBNA1-pos, LMP1-neg, EBNA2-neg) in comparison to the lymphomas infected with ΔEBNA2 alone, which have high level LMP1 expression. The gene discussed is PDLIM7; the disease is lymphoma.