The distribution of several potential contributing factors to RPL (TSH > 2.50 mU/L, parental karyotype abnormalities, acquired and hereditary thrombophilia, ANA positivity, uterine congenital abnormalities, uterine fibroids and increased resistance of the uterine arteries with PI>2.5) according to the presence or absence of adenomyosis in women with RPL is reported in Table 3. This evidence concerns the gene BTG3 and thrombophilia.