LPA and aortic valve stenosis: For example, epidemiological data of the Copenhagen City Heart Study and the Copenhagen General Population study, together comprising over 77,000 participants, confirmed a significant increase in the risk of aortic valve stenosis and myocardial infarction already at Lp(a) levels > 30 mg/dL with a threefold risk at Lp(a) levels of > 90 mg/dL [4, 5].