AGT and triple-A syndrome: Different from the peri-aortic CaPO4 injury- and Ang-II infusion-induced AAA that we used in this study, AAA lesions from combination of oral 3-aminopropionitrile (BAPN) administration and peri-aortic elastase application showed persistent aneurysm growth and more advanced-staged AAA with considerable thrombus formation and rupture, as well as higher levels of pro-inflammatory cytokines 1, 20, 48.