FMR1 and Kallmann syndrome: Not surprisingly, OB malfunctions, which include anosmia (loss of smell), hyposmia (reduced scent detection), and hyperosmia (enhanced scent detection) or OB dysplasia have been detected in patients with ASD [105–108], including patients with variants in epigenetic regulators like CBP (cyclic AMP response element binding protein binding protein; Rubinstein–Taybi syndrome risk gene) and FMR1 (fragile X messenger ribonucleoprotein 1; fragile X Syndrome risk gene) [109–111].