This is in line with the observation that hs-cTnT at 3 months, in contrast to peak hs-cTnT while hospitalized, was associated with cardiac structure and function and that chronically elevated hs-cTnT concentrations reflect permanent cardiac abnormalities in contrast to peak hs-cTnT during hospitalization which is exaggerated by the infection itself. The gene discussed is TNNT2; the disease is infection.