In LPS and polyinosinic:polycytidylic acid (POLY I:C)-induced acute lung injury (ALI)/acute respiratory distress syndrome (ARDS) models, CGA counteracts the inflammatory and oxidative stress in human airway epithelial cells and in BALB/c mice through targeting the TLR4/TLR3/NLRP3 inflammasome axis [336]. Here, NLRP3 is linked to acute respiratory distress syndrome.