Thus, DYNLT3 was found to be highly overexpressed in both BC tissues and BC cell lines, in association with N-cadherin and vimentin (VIM) overexpression associated with E-cadherin downregulation, while DYNLT3 silencing suppressed cell growth, migration and invasion via the epithelial–mesenchymal transition (EMT) and induced cell apoptosis in MDA-MB-231 and MCF7 BC cells [142]. This evidence concerns the gene VIM and breast cancer.