Considering that TGF-β is a classic inducer of epithelial–mesenchymal transition (EMT) in cells and that EMT and partial EMT of LPCs contribute to liver fibrosis and the initiation of liver cancer [12–14], we subsequently performed EMT-related experiments to confirm whether EMT occurs in LPCs after treatment with TGF-β. Here, TGFB1 is linked to Hepatic fibrosis.