To investigate the impact of Atg5 deletion on the CCL20 chemokine response of BMDMs, we selected chemokine (C-X-C motif) ligand 3 (CXCL3) [ligand of CXC chemokine receptor 2 (CXCR2)] as a control [23] based on the data that CXCR2 expression from the isolated infiltration of F4/80+ cells (macrophages) in the kidneys was not different between the MΦ atg5−/− and WT mice 4 days after severe AKI (Fig. 6E). The gene discussed is CXCR2; the disease is acute kidney injury.