SDC1 and Miyoshi myopathy: Our results demonstrated that anti-CD40 neutralizing antibody, but not the isotype control, evidently abolished the anti-MM effect of RSL3, as indicated by heavier bone lesion (Fig. 6L), increased infiltration of CD138+ plasma cells in the transplanted fetal bone (Fig. 6M), and resulted in a higher level of M protein in mouse blood (Fig. 6N).