Because EETs seem to play a role in the pathophysiology of diabetes, the aim of this study was to describe the expression profiles of EET-generating (CYP2C8, CYP2C9, CYP2J2) and EET-degrading (sEH) enzymes in HBCs from patients with T1DM and GDM compared to healthy controls. This evidence concerns the gene CYP2C8 and gestational diabetes.