Targeting tyrosine kinase receptor factor (Met) by miR-148a reduces Snail accumulation in the nucleus, promotes hepatocyte growth, activates downstream Akt Ser473 phosphorylation, and inhibits GSK-3β-Ser9 phosphorylation, thereby alleviating EMT cancer in the liver (Zhang et al., 2014). This evidence concerns the gene AKT1 and cancer.