We found obvious colocalization of STING and microglia marker IBA1 both in sham-treated group and stroke-injured group (Fig. 1D), while little or no colocalization was detected between STING and NeuN (Fig. 1H) or GFAP (Fig. 1I), indicating that STING was primarily expressed by microglia under both physiological and stroke-induced pathological conditions. This evidence concerns the gene STING1 and Stroke.