Studies have shown that treatment with sodium caprylate (medium-chain fatty acid) in rats with myocardial infarction can promote HAT KAT2A to increase the level of H3K9ac at the promoter of HO1 and NQO1 genes, improving the antioxidative stress and anti-apoptosis ability of cardiomyocytes [34]. The gene discussed is TMPRSS11D; the disease is myocardial infarction.