Therefore, according to this model, and recognizing the functional association between FLVCR1a and ALAS1, the overexpression of FLVCR1 in the cells of T2D patients may be interpreted as a cellular attempt aimed at heightening ALAS1 activity, in order to boost glucose uptake  in adipocytes, a process known to exert significant impacts on overall glucose regulation throughout the body, extending beyond its effects on adipocytes [78]. Here, FLVCR1 is linked to type 2 diabetes mellitus.