By administrating thiostrepton (TST), which is a protein translation inhibitor that has been reported to inhibit FOXM1 activity 43, we observed that pharmacological inhibition of FOXM1 was sufficient to induce tumor-inhibitory effect and potentiated the response to anti-PD-1 in the LLC mouse model (Fig. 9C to 9E). Here, FOXM1 is linked to neoplasm.