Specifically, several biologic aberrancies appear to contribute to the development of rosacea, including toll‐like receptors,26, 27 kallikrein‐related peptidase 5 and cathelicidin LL‐37,28 nuclear factor κB,29 myeloid differential factor‐88,30 and multiple genetic factors.31, 32. This evidence concerns the gene CAMP and rosacea.