MALAT1 and early-onset autosomal dominant Alzheimer disease: Peizhi and his colleagues conducted a study in an Alzheimer's disease (AD) animal model, wherein they observed that upregulating MALAT1 expression led to a reduction in neuronal apoptosis, improved neuronal functional recovery and regeneration, and a drop in levels of IL-6 and TNF-α, while concurrently boosting IL-10 level.