Most ovarian malignancies are of epithelial origin, and the dualistic model divides ovarian cancer into two types: Type I cancer, such as mucinous, endometrioid, and clear cell carcinomas are slower‐growing, less aggressive tumors with specific genetic mutations, and Type II cancer, such as high‐grade serous carcinomas and undifferentiated carcinomas, are fast‐spreading, more aggressive tumors often with p53 and BRCA gene mutations.3 Here, TP53 is linked to cancer.