The classic histopathology in DM includes a vacuolar interface dermatitis with epidermal atrophy and hyperkeratosis with mucin deposition in the dermis.[8] However, nonspecific dermatopathological findings can also be seen.[24] Given the above, it seems likely that molecular analysis of genetic markers for SAIDs may help identify SAID patients from the DM patient population, particularly in the absence of autoantibodies. Here, MUC5AC is linked to dermatomyositis.