It has been shown (Li, 2015) that salidroside inhibited the bleomycin-induced increase in serum hydroxyproline and TGF-β1, prevented the bleomycin-induced decrease in pulmonary superoxide dismutase (SOD), peroxidase (POD) and catalase (CAT), and reduced pulmonary IV collagen, malondialdehyde (MDA) and TGF-β1 content, thus improving pulmonary fibrosis in rats. The gene discussed is TGFB1; the disease is pulmonary fibrosis.