miR-30a-5p was involved in the regulation of tumor growth by controlling the expression of important oncogenes, e.g. miR-30a-5p reduced CD73 expression by targeting its 3’-UTR and inhibited lung cancer cell growth [31], and miR-30a-5p also directly interacted with YAP1 to modulate cell autophagy, invasion, and apoptosis in gastric cancer [32]. This evidence concerns the gene NT5E and neoplasm.