KRAS and Friedreich ataxia: Remarkably, within the FA, there was a focus of oncocytic proliferation that carried the same identical KRAS variant and in addition a TERTp variant; suggestive of subclonal diversification with progression into another histopathological morphology with an aggressive molecular pattern (gain of a TERTp mutation23); next to a clonally unrelated lesion (NIFTP vs FA/oncocytic proliferation).