(iv) Consequently, P-BS-CM1 → P-CM2 substantially regressed pulmonary PMN hallmarks like LOX accumulation (Supplementary Fig. 14), BMDC recruitment, S100A8 secretion (Fig. 6f), and E-cadherin expression (Supplementary Fig. 15) to levels close to healthy tumor-free mice, whereas free BS and P-BS had limited effects. The gene discussed is S100A8; the disease is neoplasm.