The upregulated genes in AM were enriched in prion-induced diseases, diabetic cardiomyopathy, phagocytosis, rheumatoid arthritis, oxidative phosphorylation, fluid shear stress-related atherosclerosis, amoebiasis, viral protein interaction with cytokines and their receptors, ECM-receptor interaction, and the IL-17 signaling pathway (Figure S4B). This evidence concerns the gene IL17A and atherosclerosis.