Next, the DEG profile of ELAVL2-high GBM patients was uploaded into IPA and the canonical pathway analysis revealed that EMT-related pathways, including wound healing signaling (z-score: -2.8) and tumor microenvironment pathways (z-score: -1.5), are highly deactivated, suggesting that ELAVL2 may repress MES-associated phenotypes in GBM (Fig. 3i). Here, ELAVL2 is linked to glioblastoma.