MV-4-11 AML cells sensitive to anti-apoptotic BCL-2 protein inhibition on “plastic” [34], cultured directly on monolayers of M- or CM-MΦ, were resistant to up to 1 μM venetoclax (Fig. 4A, B), a phenomenon we previously demonstrated [21]. The gene discussed is BCL2; the disease is acute myeloid leukemia.