Decrease in hepatic triglyceride and total cholesterol levels, decrease in gene expression related to lipogenesis and triglyceride synthesis in liver, reduction of intestinal mucus secretion (76), metabolic disorders due to alteration of intestinal microbiota (77), induction of IL-6 and IL-8 expression in gastric adenocarcinoma cells (68), induction of oxidative stress inT98G cells (63), increased AChE activity in the liver (34). Here, ACHE is linked to gastric adenocarcinoma.